Anti-Recombinant-Sarm1 antibody (1-100) [S5113MM] (STJ11105113)

SPECIFICATIONS
ClonalityMonoclonal
HostMouse
ConjugationUnconjugated
IsotypeIgG1
STJ11105113
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General Information

Short DescriptionMouse monoclonal Recombinant-Sarm1 (1-100) antibody for use in WB and ELISA in human, mouse and rat samples. Datasheet included with dilution recommendations, and related reagents.
ApplicationsWB/ELISA
HostMouse
ReactivityHuman/Mouse/Rat
NoteSTRICTLY FOR FURTHER SCIENTIFIC RESEARCH USE ONLY (RUO). MUST NOT TO BE USED IN DIAGNOSTIC OR THERAPEUTIC APPLICATIONS.

Product Properties

ClonalityMonoclonal
Clone IDS5113MM
IsotypeIgG1
ConjugationUnconjugated
ConcentrationLot specific
PurificationAffinity purification
Dilution RangeWB:1:2000-1:4000
ELISA:Recommended starting concentration is 1 Mu g/mL. Please optimize the concentration based on your specific assay requirements.
FormulationPBS with 0.05% Proclin300, 50% Glycerol, pH 7.3.
Storage InstructionStore at-20°C for up to 1 year from the date of receipt, and avoid repeat freeze-thaw cycles.

Target Information

Gene SymbolSarm1
Gene ID237868
Uniprot IDSARM1_MOUSE
Immunogen Region1-100
Immunogen SequenceMVLTLLFSAYKLCRFFTMSG PRPGADRLTVPGPDRSGGAS PWWAAGGRGSREVSPGVGTE VQGALERSLPELQQALSELK QASAARAVGAGLAEVFQLVE
SpecificityA synthetic peptide corresponding to a sequence within amino acids 1-100 of mouse Sarm1 (NP_766383.2).

Additional Info

Tissue Specificity Widely expressed in the brain and neurons (at protein level). Expressed in photoreceptor cells of the neural retina.
Post Translational Modifications Phosphorylation at Ser-548 by JNK kinases (MAPK8, MAPK9 and /or MAPK10) enhance the NAD(+) hydrolase (NADase) activity. Phosphorylation at Ser-548 and subsequent activation takes place in response to oxidative stress conditions and inhibits mitochondrial respiration.
Function NAD(+) hydrolase, which plays a key role in axonal degeneration following injury by regulating NAD(+) metabolism. Acts as a negative regulator of MYD88- and TRIF-dependent toll-like receptor signaling pathway by promoting Wallerian degeneration, an injury-induced form of programmed subcellular death which involves degeneration of an axon distal to the injury site. Wallerian degeneration is triggered by NAD(+) depletion: in response to injury, SARM1 is activated and catalyzes cleavage of NAD(+) into ADP-D-ribose (ADPR), cyclic ADPR (cADPR) and nicotinamide.NAD(+) cleavage promoting cytoskeletal degradation and axon destruction. Also able to hydrolyze NADP(+), but not other NAD(+)-related molecules. Can activate neuronal cell death in response to stress. Regulates dendritic arborization through the MAPK4-JNK pathway. Involved in innate immune response: inhibits both TICAM1/TRIF- and MYD88-dependent activation of JUN/AP-1, TRIF-dependent activation of NF-kappa-B and IRF3, and the phosphorylation of MAPK14/p38.
Protein Name Nad(+ Hydrolase Sarm1
Nadase Sarm1
Nadp(+ Hydrolase Sarm1
Sterile Alpha And Tir Motif-Containing Protein 1
Database Links Reactome: R-MMU-166166
Reactome: -MMU-936964
Reactome: -MMU-937041
Reactome: -MMU-937072
Cellular Localisation Cytoplasm
Cell Projection
Axon
Dendrite
Synapse
Mitochondrion
Associated With Microtubules
Alternative Antibody Names Anti-Nad(+ Hydrolase Sarm1 antibody
Anti-Nadase Sarm1 antibody
Anti-Nadp(+ Hydrolase Sarm1 antibody
Anti-Sterile Alpha And Tir Motif-Containing Protein 1 antibody
Anti-Sarm1 antibody
Anti-Kiaa0524 antibody

Information sourced from Uniprot.org

Citations

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